Endogenous sex steroids and risk of cervical carcinoma: results from the EPIC study.

نویسندگان

  • Sabina Rinaldi
  • Martyn Plummer
  • Carine Biessy
  • Xavier Castellsagué
  • Kim Overvad
  • Susanne Krüger Kjær
  • Anne Tjønneland
  • Françoise Clavel-Chapelon
  • Nathalie Chabbert-Buffet
  • Sylvie Mesrine
  • Annekatrin Lukanova
  • Rudolf Kaaks
  • Cornelia Weikert
  • Heiner Boeing
  • Antonia Trichopoulou
  • Pagona Lagiou
  • Dimitrios Trichopoulos
  • Domenico Palli
  • Claudia Agnoli
  • Rosario Tumino
  • Paolo Vineis
  • Salvatore Panico
  • Bas Bueno-de-Mesquita
  • Henk J van Kranen
  • Petra Hm Peeters
  • Kjersti Bakken
  • Eiliv Lund
  • Inger Torhild Gram
  • Laudina Rodríguez
  • F Xavier Bosch
  • Maria-José Sánchez
  • Miren Dorronsoro
  • Carmen Navarro
  • Aurelio Barricarte Gurrea
  • Lennart Kjellberg
  • Joakim Dillner
  • Jonas Manjer
  • Salma Butt
  • Kay-Tee Khaw
  • Nicholas Wareham
  • Naomi E Allen
  • Ruth Travis
  • Isabelle Romieu
  • Pietro Ferrari
  • Elio Riboli
  • Silvia Franceschi
چکیده

BACKGROUND Epidemiologic data and animal models suggest that, despite the predominant role of human papillomavirus infection, sex steroid hormones are also involved in the etiology of invasive cervical carcinoma (ICC). METHODS Ninety-nine ICC cases, 121 cervical intraepithelial neoplasia grade 3 (CIN3) cases and 2 control women matched with each case for center, age, menopausal status and blood collection-related variables, were identified in the European Prospective Investigation into Cancer and Nutrition (EPIC) study. Circulating levels of testosterone (T) and estradiol (E(2)); dehydroepiandrosterone sulfate (DHEAS); progesterone (premenopausal women); and sex hormone-binding globulin (SHBG) were measured using immunoassays. Levels of free (f) T and E(2) were calculated from absolute concentrations of T, E(2), and SHBG. Odds ratios (ORs) and 95% confidence intervals (CI) were computed using regularized conditional logistic regression. RESULTS Among premenopausal women, associations with ICC were observed for fT (OR for highest vs. lowest tertile = 5.16, 95% CI, 1.50-20.1). SHBG level was associated with a significant downward trend in ICC risk. T, E(2), fE(2), and DHEAS showed nonsignificant positive association with ICC. Progesterone was uninfluential. Among postmenopausal women, associations with ICC were found for T (OR = 3.14; 95% CI, 1.21-9.37), whereas E(2) and fT showed nonsignificant positive association. SHBG level was unrelated to ICC risk in postmenopausal women. No associations between any hormone and CIN3 were detected in either pre- or postmenopausal women. CONCLUSIONS Our findings suggest for the first time that T and possibly E(2) may be involved in the etiology of ICC. IMPACT The responsiveness of cervical tumors to hormone modulators is worth exploring.

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عنوان ژورنال:
  • Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology

دوره 20 12  شماره 

صفحات  -

تاریخ انتشار 2011